IgA nephropathy proteinuria treatment

IgA nephropathy in patients with proteinuria, most of the choice of hormone therapy, hormone treatment of urinary protein does have a good effect, in the short term to the protein down, or even negative. However, patients with IgA nephropathy often have encountered this situation, reduce the hormone protein will appear again and again.
In response to this problem, kidney disease hospital experts through research and a lot of practice, in the treatment of urinary protein has achieved remarkable results.

IgA Nephropathy: How to Reduce Protein in Urine

 

IgA kidney disease protein therapy for urinary protein
1, traditional Chinese medicine treatment of urinary protein. The micro-traditional Chinese medicine directly penetrate into the kidney area, so that the active ingredients of drugs into the damaged glomeruli, renal tubules, to repair, increase the glomerular protein filtration ability, improve renal tubules Of the ability of reabsorption of protein.
2, immunosuppressive therapy of urinary protein. As the active ingredient of traditional Chinese medicine treatment of urinary protein requires a process, so IgA nephropathy in the initial stage of treatment still need immunosuppressant drugs to cooperate to ensure that the protein was lowered in the near future, so that Chinese medicine penetration therapy to seize this opportunity, Repair damaged kidneys.

How to Reduce Proteinuria in IgA Nephropathy with Immunotherapy

3, if necessary, combined with hormone therapy for urinary protein. Patients treated with western medicine for IgA nephrotic urinary protein know that some patients still want to use hormones in the initial stage of treatment of urinary protein due to price and other reasons. The reason and the use of immunosuppressive agents is the same, are in order to stabilize the protein in the short term, to repair the kidney to provide a more comfortable time.
After a certain period of treatment, when the kidney tissue is the active substance of traditional Chinese medicine repair, glomerular filtration protein and renal tubular reabsorbent protein capacity will rise, gradually restored, the protein will gradually decline, and finally negative.IgA kidney disease urinary protein with the treatment
How to Reduce Proteinuria in IgA Nephropathy with Immunotherapy

 

IgA kidney disease urinary protein with the treatment include:

1, the patient in the diet to ensure high quality low protein intake, should not eat salty food, so as not to increase the burden on the kidneys, the protein filtration capacity to further decline;
2, to prevent colds, in order to minimize the probability of induced urinary protein repeated;3, should not be overworked, to maintain the amount of activity;4, in strict accordance with the doctor's advice to pay attention to every little detail in life.

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Is proteinuria a kidney failure?

Proteinuria is kidney failure Sometimes, when we urinate will find abnormal urine bubble, some people will ignore this phenomenon, some people will think that this is a very serious phenomenon. Different people have different understanding of foam urine, for those who have kidney disease, they are very concerned about the bubble urine. Because this is a typical symptom of kidney problems, but also indicates that this deteriorating renal function, if not timely treatment will lead to very serious condition.
But the bubble urine is not kidney failure, in order to better detect foam urine, you can do these tests, such as urine routine examination, urinary protein electrophoresis, 24-hour urine protein quantitative, and even urine protein.
In doing these tests, you can have a preliminary distinction between your own urine.
If the foam is large or the size is not uniform, it lasts only for a short time, this foam is normal, which is due to urinary organic and inorganic substances such as glucose and mineral salts.
If the foam is large and soon disappears, this may indicate urinary sugar substances.
If the bubble is fine and long time will not disappear, it may be proteinuria. In this case, urinary protein quantification and urinary protein electrophoresis are necessary.
In addition foam urine, if the patient has urinary urgency, frequent urination, dysuria or edema, high blood pressure or drink a lot, a lot of urine, eat a lot, so the patient recommended to the local kidney disease hospital has early diagnosis and timely and appropriate treatment The
For patients with renal failure, foam urine is very common.
Foam urine is very common in patients with renal failure, it is due to impaired glomerular filtration membrane and in some cases, renal tubules caused by excessive leakage of urinary protein.
However, when the urine bubble is reduced, this does not mean that it will improve the disease condition. Reducing the foam in the urine can also be caused by further deterioration of renal function when the glomeruli have scarring and fewer proteins can be filtered.

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Kidney disease proteinuria hazards

Nephrotic patients is a common symptom is the emergence of proteinuria, most of these symptoms occurred in patients with chronic nephritis and nephrotic syndrome patients. Protein is a necessary substance in the human body, the occurrence of proteinuria means that patients with a large number of proteins with urine discharge, affecting the health of the body. So what is the risk of proteinuria in kidney disease? See how our experts are introduced.

Kidney disease, the risk of proteinuria: proteinuria mesangial toxicity, glomerular mesangial protein accumulation will continue to lead to mesangial cells are damaged, hyperplasia of mesangial matrix synthesis increased, and ultimately cause glomerular sclerosis. And glomerular apolipoprotein A and apolipoprotein B deposition will eventually cause glomerular sclerosis.

Kidney disease, the risk of proteinuria: proteinuria can cause renal failure, mainly due to its biological changes caused by tubule cells caused by non-adaptive response. According to the current data, the protein can directly regulate the function of small tube, change its growth characteristics and its cytokines and matrix protein phenotype expression, but also the release of PDGF, FN and MCP-1 on the basal side of the tubule, induced fibrosis process.

Kidney disease, the risk of proteinuria: proteinuria toxic effects will affect the proximal tubule, increased into the renal tubular epithelial cells within the amount of protein, thereby increasing the activity of lysosomes, which means that the protein lysosome overflow into the tubules Pulp, and then the damage caused by cell damage caused by inflammation and scar generation. There may be caused by interstitial hypoxia aggravated, leading to small tube cells hypoxia, to damage.

Proteinuria will not only make a lot of protein loss, there may damage the renal tubules, glomerular. And therefore must be effective in preventing and control work, to avoid the above described kidney disease proteinuria hazards, so as not to affect your healthy life.

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Clinical manifestations of kidney disease

The most basic feature is a large number of proteinuria, hypoproteinemia, (height) edema and hyperlipidemia, the so-called "three high and one low", and other metabolic disorders characterized by a group of clinical syndrome.

1、A large number of proteinuria
A large number of proteinuria is the most important clinical manifestations of NS patients, but also the most basic pathophysiology of nephrotic syndrome. A large number of proteinuria refers to adult urine protein excretion> 3.5g / d. In normal physiological conditions, glomerular filtration membrane with molecular barrier and charge barrier, resulting in increased protein content in the original urine, when far more than the proximal tubule back to the amount of absorption, the formation of a large number of proteinuria. On this basis, where the increase in glomerular pressure and lead to high perfusion, high filtration factors (such as high blood pressure, high protein diet or a large number of infusion of plasma protein) can increase the discharge of urine protein.

2、Hypoproteinemia
Plasma albumin down to <30g / L. NS is a large number of albumin lost from the urine, promote albumin liver compensatory synthesis and increased renal tubular decomposition. Low albuminemia occurs when liver albumin synthesis is not sufficient to overcome loss and breakdown. In addition, NS patients due to gastrointestinal mucosal edema caused by eating loss, lack of protein intake, poor absorption or loss, but also increase the causes of hypoalbuminemia.
In addition to plasma albumin reduction, some plasma immunoglobulin (such as IgG) and complement components, anticoagulant and fibrinolytic factors, metal binding protein and endocrine binding protein can also be reduced, especially a large number of proteinuria, glomerular Pathological damage is more severe and non-selective proteinuria is more significant. Patients prone to infection, hypercoagulable, lack of trace elements, endocrine disorders and immune dysfunction and other complications.

3. edema
NS hypoalbuminemia, plasma colloid osmotic pressure de
creased, so that water from the vascular cavity into the tissue gap, is the basic cause of NS edema. Recent studies have shown that about 50% of patients with normal or increased blood volume, plasma renin levels normal or decreased, suggesting that some of the primary renal sodium, water retention factors in the NS edema mechanism play a role.

4. Hyperlipidemia
The reason why NS is associated with hyperlipidemia is not yet fully elucidated. High cholesterol and / or hypertriglyceridemia, serum LDL, VLDL and lipoprotein (α) concentration increased, often associated with hypoproteinemia. Hypercholesterolemia is mainly due to increased liver lipoprotein production, but in the surrounding cycle to reduce the decomposition also play a part of the role. Hypertriglyceridemia is mainly due to catabolic disorders caused by increased liver synthesis as a secondary factor.

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