chronic nephritis symptoms

Nephritis is a multiple nephropathy, chronic nephritis is the development of acute nephritis disease progression. Chronic nephritis early symptoms are more concealed, easily overlooked by patients. To understand the early symptoms of chronic nephritis can be found in a timely manner, then chronic kidney disease posted what early symptoms? The following is a detailed answer to the relevant experts.

One: eyelid edema
Chronic nephritis early prone to eyelid edema is the main reason: the kidneys on the water excretion and regulation of damage, so that children in the body of water and sodium increased, too much water in the body loose tissue, and eyelids are loose tissue More parts. Eyelid edema is characterized by the morning to get up when the activity is diminished.

Two: hematuria
Hematuria with gross hematuria and microscopic hematuria of the points, which also belong to the early symptoms of chronic nephritis. Gross hematuria is the naked eye can see the hematuria, urine was washed meat samples, turbid and red, and some children with blood in the blood clots or blood clots; and microscopic hematuria is only observed under the microscope can be found, The number of red blood cells per high power field is greater than one. Hematuria is also the majority of children with chronic nephritis early symptoms. Of course, hematuria for many reasons, nephritis is just one of them. Therefore, when parents find children with unexplained hematuria, should be timely medical treatment. According to the doctor's advice to do the relevant checks, if necessary, do routine urine examination to rule out the possibility of renal hematuria.

Three: increase in urine foam
Urine foam increased to a smaller bubble-based, and interconnected together, a long time can not be dispersed, suggesting that children with urine protein, caused by higher tension. This is also one of the early symptoms of chronic nephritis. Of course, the accuracy of this symptom is poor. If you do not have a certain medical knowledge, often by parents or children ignored. The easiest way is to go to the hospital to check the urine to rule out the possibility of proteinuria. Because the earliest changes in chronic nephritis are urine.

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Traditional Chinese medicine chronic kidney disease

Western medicine on the treatment of chronic nephritis are not satisfactory place, some people began to think of traditional Chinese medicine treatment. We all know that traditional Chinese medicine treatment of chronic nephritis and Western medicine are very different, let us introduce you to the following.

With the aging of the population and changes in people's lifestyles, the traditional infectious diseases are decreasing, chronic immune diseases are becoming the first factor threatening human health. Relative to other chronic diseases, chronic kidney disease has become one of the most common diseases that threaten human health. Suffering from chronic kidney disease, if not satisfied with the control will eventually enter the chronic renal insufficiency and uremia.

In the treatment of traditional Chinese medicine treatment of chronic nephritis have a unique view. Micro-traditional Chinese medicine infiltration therapy for chronic nephritis is a new breakthrough in the treatment of chronic nephritis, Chinese herbal medicine through the regulation of systemic immune function, repair glomerular basement membrane, the active ingredients of drugs through the penetration of points to the patient's kidney, , Blood circulation, reduced turbidity detoxification as a way to clear the kidneys, activate the function of the kidney for the purpose.

Chinese medicine treatment of chronic nephritis to promote and improve the destruction of necrotic kidney microcirculation, accelerate the kidney metabolism, so that the active ingredients in the drug to play a full role in repair damaged glomerular, enhanced glomerular reabsorption, restore normal renal physiological function , From the fundamental treatment of chronic nephritis.

Chronic nephritis is a syndrome caused by multiple causes of glomerular lesions. It is not the true sense of inflammation, but a systemic disease. At present, the etiology of chronic nephritis is not clear. It is certain that infection may cause acute episodes of chronic nephritis, such as urinary tract infections, respiratory infections, and the like. Clinical manifestations of edema, hypertension, abnormal urine and other symptoms. For the treatment of chronic nephritis, simply to eliminate proteinuria, hematuria as the goal, fail to achieve a real effective treatment. The treatment of chronic nephritis is more focused on the treatment of progressive renal dysfunction, so the need for comprehensive treatment measures.

Chronic nephritis is due to the inherent renal cell injury can not be a long-term treatment of systemic norms, dysfunction, can not stop the leakage of protein and red blood cells, extracellular matrix accumulation, start renal fibrosis, and renal fibrosis and further aggravated renal cell damage The The treatment of chronic nephritis can not simply eliminate blood, eliminate protein, to fundamentally block the process of renal fibrosis, repair pathological damage, and strive to have damaged cell function recovery.

Chinese medicine treatment of chronic nephritis we all know, if you would like to learn more about traditional Chinese medicine treatment of chronic nephritis

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kindeyl failure

1. To the cardiovascular aspects of the harm: If the patient suffers from severe renal insufficiency, will lead to vascular calcium, hypertension, arrhythmia, pericarditis, heart failure, myocardial damage, hardening of the arteries.The following are the same as the "

2. To the hematopoietic aspects of the harm: severe renal insufficiency patients one of the most obvious symptoms is severe anemia. Disease development to the late when there may be bleeding tendency, more common symptoms of epistaxis, ecchymosis.

3. To the respiratory side of the harm: renal insufficiency in the late when there may be pleurisy, uremic bronchitis, pneumonia and so on. The following are the same as the "The following are the same as the "

4. To the digestion of the hazards: renal insufficiency patients will be diarrhea, mouth urine smell, vomiting, nausea and other symptoms, serious cases may occur in the case of gastrointestinal bleeding.The following are the same as the "

5. To the mental and neurological aspects of the harm: the spirit of severe renal insufficiency patients is not very good, and will feel the body without any strength, will feel dizziness, headache, memory will be worse than before, often sleep at night Do not feel, his hands and feet numbness of the situation, there will be convulsions, coma, hand, foot incest, delirium, muscle fibrillation and other symptoms.

6. Harm to the skin: The skin of patients with severe renal insufficiency appears to be luster and the skin will dry and desquamate.

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kidney cysts Causes

kidney cysts in the absence of the corresponding cause of kidney cysts, kidney cysts according to their clinical manifestations can be attributed to "hematuria", "low back pain", "accumulation", "virtual" and other diseases. "Lingshu. The dirty chapter "that" kidney is good disease low back pain, easy to hurt with evil. " And "Lingshu. Swelling of the article "and" kidney swelling "of the records, such as" kidney swelling, abdominal full lead back to the central heart, waist pain "and other descriptions, and kidney cysts are similar to the characteristics of the main card.

Summary of historical literature, combined with the clinical features of the cause of kidney cysts, experts believe that the cause of kidney cysts and kidney essence deficiency, poor blood, phlegm endogenous, vein silt resistance related to a simple surgical treatment palliative, Fundamentally inhibit the increase in cysts, it is best to use integrated traditional Chinese and Western medicine treatment, through Huoxuetongluo, stasis removal and other effects, so that natural reduction of kidney cysts. The specific cause and process of kidney cysts are as follows:

1, simple kidney cysts cause

Simple kidney cysts may be derived from immature glomerular development in the retention of cysts, or inflammation, ischemia caused by the results of obstruction of the pipeline system.

2, polycystic kidney disease causes

Polycystic kidney system kidney cortex, medulla appear numerous cases of a hereditary disease, when the cyst is small when the renal parenchyma does not cause any threat, but the cyst with the patient's age gradually increased, in the process of increasing Due to cysts oppression of renal tissue and renal blood vessels, leading to local microcirculation of renal tissue disorders, so that the kidney ischemia, hypoxia, resulting in renal capillary endothelial cell damage, and to attract blood circulation in inflammatory cell infiltration, Produce and aggravate the glomerular local inflammatory response, initiated the renal fibrosis. At this point if not timely and effective treatment control, damaged tissue cells will release a series of renal toxicity factor, making the kidney cells in phenotype transformation, accelerated renal function damage rate, especially in the renal interstitial Fibroblasts transformed into myofibroblasts after the secretion of a large number of non - easily degradable extracellular matrix and immune complexes, due to the synthesis and degradation of the extracellular matrix of the dynamic equilibrium imbalance, a large number of extracellular matrix accumulation, resulting in glomerular Sclerosis, tubular atrophy, resulting in renal fibrosis gradually replaced the healthy kidney cells, kidney function gradually lost, the clinical development of renal end stage, patients with renal failure, that uremic period.

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Your time is precious and so is your kidney health. That's why Our Kidney Disease Clinic is offering various services online. Through our four parts--KIDNEY DISEASES, OUR TREATMENT, SYMPTOMS A~Z AND TEST, you can get a full understanding of your kidney condition.

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Kidney disease proteinuria hazards

Nephrotic patients is a common symptom is the emergence of proteinuria, most of these symptoms occurred in patients with chronic nephritis and nephrotic syndrome patients. Protein is a necessary substance in the human body, the occurrence of proteinuria means that patients with a large number of proteins with urine discharge, affecting the health of the body. So what is the risk of proteinuria in kidney disease? See how our experts are introduced.

Kidney disease, the risk of proteinuria: proteinuria mesangial toxicity, glomerular mesangial protein accumulation will continue to lead to mesangial cells are damaged, hyperplasia of mesangial matrix synthesis increased, and ultimately cause glomerular sclerosis. And glomerular apolipoprotein A and apolipoprotein B deposition will eventually cause glomerular sclerosis.

Kidney disease, the risk of proteinuria: proteinuria can cause renal failure, mainly due to its biological changes caused by tubule cells caused by non-adaptive response. According to the current data, the protein can directly regulate the function of small tube, change its growth characteristics and its cytokines and matrix protein phenotype expression, but also the release of PDGF, FN and MCP-1 on the basal side of the tubule, induced fibrosis process.

Kidney disease, the risk of proteinuria: proteinuria toxic effects will affect the proximal tubule, increased into the renal tubular epithelial cells within the amount of protein, thereby increasing the activity of lysosomes, which means that the protein lysosome overflow into the tubules Pulp, and then the damage caused by cell damage caused by inflammation and scar generation. There may be caused by interstitial hypoxia aggravated, leading to small tube cells hypoxia, to damage.

Proteinuria will not only make a lot of protein loss, there may damage the renal tubules, glomerular. And therefore must be effective in preventing and control work, to avoid the above described kidney disease proteinuria hazards, so as not to affect your healthy life.

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Affect the prognosis of chronic renal failure

Renal failure can be divided into acute and chronic, acute renal failure disease progression is rapid, usually due to kidney blood supply (such as trauma or burns), the kidneys caused by a certain cause of damage caused by damage or poison damage caused by Acute renal failure. And chronic renal failure, also known as chronic renal failure, is caused by a variety of chronic diseases, kidney damage and progressive deterioration of the results, so that the body in the excretion of metabolic waste and regulation of water, electrolyte, acid-base balance and other aspects of the clinical syndrome group The Is one of the important diseases that threaten life. The end of chronic renal failure, often referred to as uremia. Factors affecting the prognosis of chronic renal failure, the following are more common:

(1) primary renal disease itself is closely related to the prognosis of chronic renal failure.

(2) various infections are the most common factors affecting the prognosis of chronic renal failure, of which the respiratory and urinary tract infections are the most common.

(3) urinary tract obstruction is also a common prognostic factor, and often coexist with urinary tract infection.

(4) the use of nephrotoxic drugs.

(5) causes a variety of causes of renal insufficiency, such as severe vomiting, diarrhea caused by shock, low blood pressure, long-term excessive use of diuretics and so on.

(6) heart failure caused by various causes.

(7) renal hypertension on renal function has a direct damage.

(8) water and electrolyte disorders and so on.

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Differential diagnosis of kidney disease

NS complications are important factors affecting the long-term prognosis of patients should be actively prevention and treatment.

1. infection

Usually in the hormone treatment without the use of antibiotics to prevent infection, or not only fail to prevent the purpose, but may induce fungal infection. Once found infection, should be timely selection of pathogenic bacteria sensitive, potent and no renal toxicity of active treatment of antibiotics, a clear infection should be removed as soon as possible. Severe infection is difficult to control should be considered to reduce or disable the hormone, but depends on the specific circumstances of the patient.

2. Thrombosis and embolism complications

It is generally believed that when plasma albumin is less than 20 g / L (idiopathic membranous nephropathy less than 25 g / L) anticoagulant therapy can be given heparin (also available low molecular weight heparin) subcutaneous injection or oral warfarin. Anticoagulation at the same time can be supplemented by antiplatelet drugs, such as dipyridamole or aspirin orally. On the occurrence of thrombosis, embolism should be as early as possible (6 hours the best results, but still effective within 3 days) to give urokinase or streptokinase systemic or local thrombolysis, combined with anticoagulant therapy, anticoagulants should generally continue to use More than six months. Anticoagulation and thrombolytic therapy should avoid excessive drug lead to bleeding.

3. acute renal failure

NS complicated with acute renal failure such as improper handling can be life-threatening, if given timely treatment, the majority of patients is expected to recover. The following measures can be taken:

(1) loop diuretics on loop diuretics are still effective should be a larger dose to wash the blocked tubular tubules;
(2) hemodialysis diuretic ineffective, and has reached the dialysis indications, should be hemodialysis to maintain life, and after the appropriate addition of plasma products dehydration to reduce renal interstitial edema;
(3) the original disease treatment because of its pathological type mostly small lesions of kidney disease, should be actively treated;
(4) alkaline urine can be oral sodium bicarbonate alkaline urine, in order to reduce tube formation.

4. Protein and fat metabolism disorders

It is often difficult to correct metabolic disorders before NS remission, but should adjust the amount and structure of protein and fat in the diet, and strive to minimize the impact of metabolic disorders. At present, many drugs can be used to treat protein and fat metabolism disorders. Such as: ACEI and angiotensin Ⅱ receptor antagonists can reduce urinary protein; studies have suggested that traditional Chinese medicine Astragalus can promote liver albumin synthesis, and may also reduce the role of hyperlipidemia. (HMG-CoA) reductase inhibitors, such as lovastatin statins, or lower triglyceride-based clofibrate Class, such as fenofibrate and so on. NS remission after hyperlipidemia can be natural relief, you do not need to continue drug treatment.

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Clinical manifestations of kidney disease

The most basic feature is a large number of proteinuria, hypoproteinemia, (height) edema and hyperlipidemia, the so-called "three high and one low", and other metabolic disorders characterized by a group of clinical syndrome.

1、A large number of proteinuria
A large number of proteinuria is the most important clinical manifestations of NS patients, but also the most basic pathophysiology of nephrotic syndrome. A large number of proteinuria refers to adult urine protein excretion> 3.5g / d. In normal physiological conditions, glomerular filtration membrane with molecular barrier and charge barrier, resulting in increased protein content in the original urine, when far more than the proximal tubule back to the amount of absorption, the formation of a large number of proteinuria. On this basis, where the increase in glomerular pressure and lead to high perfusion, high filtration factors (such as high blood pressure, high protein diet or a large number of infusion of plasma protein) can increase the discharge of urine protein.

2、Hypoproteinemia
Plasma albumin down to <30g / L. NS is a large number of albumin lost from the urine, promote albumin liver compensatory synthesis and increased renal tubular decomposition. Low albuminemia occurs when liver albumin synthesis is not sufficient to overcome loss and breakdown. In addition, NS patients due to gastrointestinal mucosal edema caused by eating loss, lack of protein intake, poor absorption or loss, but also increase the causes of hypoalbuminemia.
In addition to plasma albumin reduction, some plasma immunoglobulin (such as IgG) and complement components, anticoagulant and fibrinolytic factors, metal binding protein and endocrine binding protein can also be reduced, especially a large number of proteinuria, glomerular Pathological damage is more severe and non-selective proteinuria is more significant. Patients prone to infection, hypercoagulable, lack of trace elements, endocrine disorders and immune dysfunction and other complications.

3. edema
NS hypoalbuminemia, plasma colloid osmotic pressure de
creased, so that water from the vascular cavity into the tissue gap, is the basic cause of NS edema. Recent studies have shown that about 50% of patients with normal or increased blood volume, plasma renin levels normal or decreased, suggesting that some of the primary renal sodium, water retention factors in the NS edema mechanism play a role.

4. Hyperlipidemia
The reason why NS is associated with hyperlipidemia is not yet fully elucidated. High cholesterol and / or hypertriglyceridemia, serum LDL, VLDL and lipoprotein (α) concentration increased, often associated with hypoproteinemia. Hypercholesterolemia is mainly due to increased liver lipoprotein production, but in the surrounding cycle to reduce the decomposition also play a part of the role. Hypertriglyceridemia is mainly due to catabolic disorders caused by increased liver synthesis as a secondary factor.

Understanding Kidney Disease
Your time is precious and so is your kidney health. That's why Our Kidney Disease Clinic is offering various services online. Through our four parts--KIDNEY DISEASES, OUR TREATMENT, SYMPTOMS A~Z AND TEST, you can get a full understanding of your kidney condition.

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