Nursing of lupus nephritis

   As lupus erythematosus patients with long-term use of glucocorticoids, easy to cause steroid diabetes and Cushing syndrome, it is appropriate to control appetite, eat less high sugar content of food. Lupus nephritis patients should be more calcium supplement to prevent glucocorticoid caused by osteoporosis; eat more foods rich in vitamins and fruits.
Lupus Nephritis: Basics, Diagnosis, Symptoms and Treatment

   Lupus nephritis patients should not eat or eat less food with enhanced light sensitivity: such as figs, vetch, rape, yellow pepper and celery, such as eating should avoid sun exposure. Mushrooms, mushrooms and other mushrooms and certain food dyes and tobacco can also induce systemic lupus erythematosus potential role, but also try not to eat or eat less.

Lupus with Creatinine 121: Does It Mean I Have Kidney Failure

   Patients with lupus nephritis should be associated with high protein diet with renal damage in patients with systemic lupus erythematosus often have a large number of protein loss from the urine, can cause hypoproteinemia, it must add enough high-quality protein, drink milk, eat more beans Products, eggs, lean meat, fish and other protein-rich foods.

Home Remedies for Lowering Creatinine Level 4.1 with Lupus Nephritis
 No matter what is the causes, kidney disease has affected the normal life of patients gradually. I hope our medical group can do our best to help the kidney disease patients all over the world. If you want to get some treatment or diet suggestions for kidney problem, you can feel to contact me by E-mail renal-disease@hotmail.com, 
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Prevention of lupus nephritis

Lupus erythematosus is an autoimmune disease caused by a problem with the autoimmune system, and the disease that is present in this disorder is very difficult for the treatment of such diseases, so for us, Do the prevention of lupus erythematosus nephritis?

(1) attention to physical factors
Systemic lupus erythematosus close relatives of the incidence rate of up to 5% to 12%, the same egg incidence of up to 69%, lupus patients relatives of other autoimmune diseases such as rheumatism, dermatomyositis, scleroderma, dryness and psoriasis and other morbidity Also high, black and Asian descendants are higher than those of Caucasians, all of which suggest that lupus is affected by genetic factors. And female multiple, patients with low levels of testosterone, then the endocrine factors, especially estrogen in patients with lupus in vivo and the incidence of lupus nephritis. Therefore, for the basis of autoimmune disease in patients with lupus erythematosus, including relatives of autoimmune disease, should be highly vigilant, once sick should think of their own immune disease, once suffering from autoimmune diseases, should be actively treated, To prevent lupus nephritis caused by kidney damage.

(2) pay attention to drug toxicity
Lupus erythematosus-related drugs are hydralazine, procaine amide, isoniazid, methyldopa, cloprazine and quinidine, especially before the two are common. May be related to the drug in the hydrazine, amine, mercapto groups. Therefore, patients with lupus erythematosus-based lupus erythematosus should pay attention to the toxicity of these drugs to patients with lupus erythematosus.

(3) to avoid sun exposure
Ultraviolet radiation aggravated lupus nephritis is more common. Because the UV can make DNA into thymine dimer, and the antigenic enhancement, to promote systemic lupus erythematosus occurred. Therefore, patients with lupus nephritis in their daily lives should be avoided in the strong sunlight for a long time exposure to reduce the excessive UV radiation caused by renal damage.
(4) active treatment of viral infection

Viral infection may be related to the occurrence of systemic lupus erythematosus. Therefore, we should actively treat a variety of viral infections. Especially for upper respiratory tract infection such a "minor illness", must not be taken lightly. Because the "wind for the long illness", many serious illness is the cold and the beginning of the disease and thus harm the crowd.

Your time is precious and so is your kidney health.

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our doctor will reply you  with professional suggestion.

Lupus nephritis etiology

The etiology of SLE and lupus nephritis (LN) is not yet fully defined. Mainly due to a number of factors, such as genetic, viral infection, immune abnormalities, sunlight or ultraviolet radiation, some drugs induced and estrogen and other autoimmune inflammatory diseases. Its induced kidney disease (ie, LN) is typical of autoimmune complex nephritis.
     1. immune genetic defects
SLE and genetic factors related to the incidence of the family as high as 3% to 12%, there is a clear tendency to gather the family. In the SLE patients with a wide range of studies found that SLE closely related genes found in some of the HLA gene sites, especially the HLA DR region, HLA phenotype was polymorphic. Studies have shown that haploid HLA B8 / DR2 in the population is more likely to produce hypersensitivity to cellular and humoral reactions; this may be due to the presence of HLA-encoded polymorphisms in T and B lymphocytes and antigens For T inhibition of cell dysfunction, autoantibodies and globulin increased. It is now believed that the SLE susceptibility gene is a gene of different structures of T cell antigen receptors. Recent studies have found that some of the T cell B chain polypeptide structure and HLA DR in the same individual at the same time, improve the possibility of the pathogenesis of SLE. In addition, SLE also has a variety of complement defects, such as C2, C1q, C1r, C1s, C4, C5, C8 and Bf, TNF, C1 esterase inhibitor deficiency. These complement components or genetic defects, can affect the traditional complement activation pathway, increase the body's sensitivity to infection and other factors and SLE susceptibility related.

2. external environmental factors
Induced or aggravated SLE many external factors, including chronic infection, drugs, physical factors, emotional stimuli, living environment and so are more important.
Commonly known as chronic viral infection, under the electron microscope found in patients with SLE patients with tube-like inclusion bodies, and vice mucin nuclear protein and core tubular structure similar, but further study that this is a cell damage non-specific which performed. It was also found in SLE patients with glomerular endothelial cell plasma, vascular endothelial cells, lesions were found similar inclusion body material. But the inclusion of body-like substances from the organization has not been able to separate the virus, so the relationship between these substances and virus infection to be confirmed. In SLE patients, there are a variety of high titer antiviral antibodies, such as anti-measles, anti-rubella, anti-parainfluenza, anti-EB virus, anti-epidemic mumps, anti-stick virus and other antibodies. In patients with serum ds-DNA, dsRNA and RNA-DNA and other retrovirus antibodies. It has also been suggested that the incidence of SLE and C-type RNA virus is closely related. In general, there are many indications that viral infection may be one of the causes of SLE, but has not yet confirmed the virus infection and SLE patients with immune regulation abnormalities and autoimmune. In addition, some people think that the incidence of SLE and tuberculosis or streptococcal infection.

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