The pathogenesis of polycystic kidney disease

Polycystic kidney kidney parenchyma in the size of the cysts, large can be large, small can only be visible to the naked eye, so that the whole increase in renal volume, the surface was rugged cystic processes, Yellow syrup, sometimes dark brown or reddish brown due to bleeding. There are two types of polycystic kidney disease, autosomal recessive genotype (infant type) polycystic kidney disease and autosomal dominant genotype (adult type) polycystic kidney disease.
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As a hereditary nephropathy, polycystic kidney disease, the main cause of the disease is the existence of abnormal chromosomal genes. More than 90% of polycystic kidney disease in patients with abnormal gene located on the 16th chromosome. From the patient's fetal period, this abnormal situation has been formed and persistence, the vast majority of patients with polycystic kidney disease to 40 years old may be clinical symptoms. Nephrotic patients begin to originate from the renal tubular parts of the kidney, including proximal tubules and distal tubules. Origin of different parts,

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Kidney capsule fluid properties will also be different: originated in the proximal tubules, cyst fluid components such as Na +, K +, CI-, H +, creatinine, urea and other components within the plasma similar; originated in the distal tube, Liquid Na +, K + concentration is low, CI-, H +, creatinine, urea and other high concentrations.
From the pathological point of view, the formation of polycystic kidney disease, and the following three factors are inseparable:

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1, renal pelvic epithelial cell abnormal proliferation: polycystic kidney disease in patients with renal pelvic epithelial cells in a mature incomplete or re-development state, which is the development of mature bacteria due to obstacles, so that cells in the An immature state, which shows cell proliferation.
2, renal cell migration abnormalities: the performance of cell transport is closely related to Na + -K + -ATP ase subunit combination, distribution and activation of the expression changes; cell signaling abnormalities and changes in ion transport channels.
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3, extracellular matrix abnormal proliferation.
As the polycystic kidney disease in patients with abnormal genes, gene ischemia led to changes in renal cell growth and renal interstitial abnormalities. With the recurrence of nephropathy in patients with polycystic kidney disease, renal cell abnormalities and abnormal deposition of extracellular matrix, will make the kidneys fibrosis, sclerosis, atrophic degeneration, kidney disease will also be gradually from the initial polycystic kidney Become kidney failure, uremia, so that patients with polycystic kidney disease to bear more pain. Therefore, the patients with polycystic kidney disease, has repeatedly stressed that once the symptoms of polycystic kidney disease must be timely medical treatment, so as not to delay the best treatment time.

No matter what is the causes, kidney disease has affected the normal life of patients gradually.I hope our medical group can do our best to help the kidney disease patients all over the world. If you want to get some treatment or diet suggestions for kidney problem, you can feel to contact me by E-mail renal-disease@hotmail.com,or WhatsApp / Phone:008613633219293.

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