Prevention of renal failure in daily life

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Prevention of infection - a large part of the causes of kidney failure is due to infection-induced, so to prevent kidney failure must pay attention to prevent infection, such as cold, rheumatism, wind-heat, hot heat, hot and so may lead to kidney failure, To avoid sweating when the wind, rainy days out, wearing wet clothes and so on.

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Enhance physical fitness - to avoid kidney failure to protect kidney health will often exercise the body to enhance physical fitness, exercise a variety of ways, are conducive to enhance physical fitness, improve the body resistance to prevent infection after the bacterial virus immune response occurred.

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Caution with nephrotoxic drugs - some drugs on the kidney damage is great, so we should be vigilant to avoid taking. The above is for everyone to introduce the method of preventing renal failure, if in accordance with the above method of prevention, we can be a good protection of our kidneys, to avoid kidney damage, which effectively prevent the occurrence of renal failure.

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The pathogenesis of renal failure

The pathogenesis of the disease complex, is not yet fully understood.
1. Health nephron unit theory
Various causes of renal parenchymal disease, leading to most of the kidney unit damage. Residual small part of the normal damage to the function of the normal kidney unit, due to the normal needs of the body to compensate for the doubled work. Resulting in "health" of renal units in the compensatory hypertrophy, glomerular filtration function and renal tubular processing filtrate function enhancement, and ultimately lead to glomerular sclerosis and loss of function. Finally, "health" nephron gradually reduced, gradually diminished renal function.

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2. uremic toxins doctrine
Uremic toxins, may be renal failure accumulated in the body of a variety of substances, including PTH, phosphorus, urea, creatinine, guanidine, phenols and indole and so on.
These substances have the following characteristics:
① body concentration content higher than normal.
② high concentration and specific symptoms of uremia.
③ can be carried out chemical identification and quantitative determination.
④ substance concentration and uremic patients with similar concentrations of body fluids, the emergence of similar toxic effects.

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3. The theory of imbalance
Renal insufficiency when the body showed a series of pathological symptoms, the body for the corresponding adjustments to correct, but the compensatory change but led to a new imbalance, that imbalance, and thus produce a series of clinical symptoms. Such as phosphorus metabolism.

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4. Glomerular hypertension and compensatory hypertrophy theory
Health neonatal unit of the small arterial resistance decreased, and the ball out of small arterial resistance increased. Leading to high pressure in the glomeruli, high perfusion and high filtration. Glomerular hypertension to thick arterial wall thickening and capillary wall tension increased, causing ischemia and endothelial cell damage, mesangial cells and matrix hyperplasia, to promote the residual glomerular compensatory hypertrophy, glomerular sclerosis, renal function Further deteriorated.
5. Tubular hyperglycemia
Residual renal units of the renal tubules, especially proximal renal tubules, the metabolism of hyperthyroidism, oxygen free radicals increased, cell damage, so that tubulointerstitial lesions continued, renal unit function loss.

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Renal insufficiency

    Chronic Kidney Failure   (CRF) refers to a variety of kidney disease caused by slow progressive renal damage, and finally lead to complete loss of uremia and renal function, causing a series of clinical symptoms and biochemical, endocrine and other metabolic disorders Clinical syndrome. From the onset of primary disease to the beginning of renal insufficiency, the interval can be several years to more than ten years. Chronic renal failure is a serious stage of renal insufficiency.

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Causes of renal insufficiencyFirst, the cause
① infectious nephropathy: such as chronic pyelonephritis, kidney tuberculosis, etc .;② chronic glomerulonephritis: such as IgA nephropathy, membranoproliferative glomerulonephritis, focal segmental sclerosing glomerulonephritis and mesangial proliferative glomerulonephritis;

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③ toxic kidney disease: such as heavy metal toxic kidney disease, analgesic nephropathy;④ vascular renal disease: such as renal vascular hypertension, hypertension, renal arteriosclerosis and so on;⑤ hereditary nephropathy: such as polycystic kidney disease, Alport syndrome;⑥ systemic systemic diseases, such as vasculitis kidney damage, multiple myeloma, lupus nephritis;

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⑦ metabolic abnormalities caused by kidney damage: such as diabetic nephropathy, amyloidosis kidney disease and gouty kidney disease;⑧ obstructive nephropathy: such as ureteral obstruction, reflux kidney disease, urinary tract stones and so on.